| Acetylcholine (32) |  |
| Dopamine (52) | |
| Gaba (23) | |
| Neurotransmitter (83) | |
| Serotonin (47) | |
| Acetylcholine (32) | |
| Dopamine (52) | |
| Gaba (23) | |
| Neurotransmitter (83) | |
| Serotonin (47) | |
the ocial neuronsare inhibitory neurons (which release the neurotransmitter GABA or gamma aminobutyric-acid acid) while the elf-grooming neuronsare excitatory neurons
and inhibitory (GABA) signals to the VTA. But contrary to expectation, it was the inhibitory signals, not the excitatory ones,
When GABA projections alone were activated, the mice behaved bizarrely, gnawing on the bottom of the cage
-and GABA-producing neurons in the VTA. The lab is now investigating how feeding and sucrose-seeking behaviors differ based on the target neuron type.
-aminobutyric acid (GABA), a naturally produced charged molecule used by the body to control pain, to the precise spot where an injured nerve makes contact with the spinal cord.
They then injected GABA, a compound naturally produced in the brain and that inhibits neurons,
Arrayarraytwo amino acids, glutamate and GABA, are the yin and yang of the brain, directing its emotional tides
GABA, on the other hand, calms us and helps us sleep, blocking the action of the excitable glutamate.
benzodiazepine, activates GABA receptors in our brains. There are two kinds of GABA receptors. One kind, synaptic GABA receptors, works in tandem with glutamate receptors to balance the excitation of the brain in response to external events such as stress.
The other population extra-synaptic GABA receptors, are independent agents. They ignore the peppy glutamate.
Instead, their job is focused internally, adjusting brain waves and mental states according to the levels of internal chemicals, such as GABA, sex hormones and micro RNAS.
Extra-synaptic GABA receptors change the brain's state to make us aroused, sleepy, alert, sedated, inebriated or even psychotic.
However, Northwestern scientists discovered another critical role; these receptors also help encode memories of a fear-inducing event
and then store them away, hidden from consciousness.""The brain functions in different states, much like a radio operates at AM and FM frequency bands,
when these extra-synaptic GABA receptors are activated, the memory of this event cannot be accessed unless these receptors are activated once again,
a drug that stimulates extra-synaptic GABA receptors.""It's like we got them a little inebriated,
when the extra-synaptic GABA receptors were activated with the drug, they changed the way the stressful event was encoded.
activate the extra-synaptic GABA system and form inaccessible traumatic memories. Arraymemories are stored usually in distributed brain networks including the cortex
They then injected GABA, a compound naturally produced in the brain and that inhibits neurons,
Dr. Thompson and his team focused on another neurotransmitter besides serotonin, an inhibitory compound called GABA.
they examined a class of compounds that reduce the inhibitory messages sent via GABA. They predicted that these compounds would restore excitatory strength.
called GABA-NAMS, minimize unwanted side effects because they are precise: they work only in the parts of the brain that are essential for mood.
Giving stressed rats GABA-NAMS successfully reversed experimental signs of a key symptom of depression, anhedonia,
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